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GABA Neuronal Deletion of Shank3 Exons 14-16 in Mice Suppresses Striatal Excitatory Synaptic Input and Induces Social and Locomotor Abnormalities Yoo, Taesun; Cho, Heejin; Lee, Jiseok; Park, Haram; Yoo, Ye Eun; Yang, Esther; Kim, Jin Yong; et al, FRONTIERS IN CELLULAR NEUROSCIENCE, v.12, no.341, 2018-10 |
Shank3 Exons 14-16 Deletion in Glutamatergic Neurons Leads to Social and Repetitive Behavioral Deficits Associated With Increased Cortical Layer 2/3 Neuronal Excitability![]() Yoo, Taesun; Cho, Heejin; Park, Haram; Lee, Jiseok; Kim, Eunjoon, FRONTIERS IN CELLULAR NEUROSCIENCE, v.13, 2019-10 |
Suppression of NMDA receptor function in mice prenatally exposed to valproic acid improves social deficits and repetitive behaviors![]() Kang, Jaeseung; Kim, Eunjoon, FRONTIERS IN MOLECULAR NEUROSCIENCE, v.8, 2015-05 |
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