DC Field | Value | Language |
---|---|---|
dc.contributor.author | 김은준 | ko |
dc.contributor.author | 강봉균 | ko |
dc.contributor.author | 이민구 | ko |
dc.date.accessioned | 2017-12-20T02:10:28Z | - |
dc.date.available | 2017-12-20T02:10:28Z | - |
dc.date.issued | 2016-01-12 | - |
dc.identifier.uri | http://hdl.handle.net/10203/230323 | - |
dc.description.abstract | The present disclosure relates to an ASD genetically engineered model carrying a deletion of Shank2 gene and having reduced NMDA receptor function. According to the present disclosure, genetically engineered mice that show the clinical features of ASD due to the deletion of the Shank2 gene can be obtained, and the genetically engineered mice can be effectively used to screen candidate therapeutic agents. | - |
dc.title | Genetically engineered mouse model for autism spectrum disorder having deletion of Shank2 gene and use thereof | - |
dc.title.alternative | Shank2 유전자가 결실된 자폐증 모델 형질전환마우스 및 그 용도 | - |
dc.type | Patent | - |
dc.type.rims | PAT | - |
dc.contributor.localauthor | 김은준 | - |
dc.contributor.nonIdAuthor | 강봉균 | - |
dc.contributor.nonIdAuthor | 이민구 | - |
dc.contributor.assignee | KAIST | - |
dc.identifier.iprsType | 특허 | - |
dc.identifier.patentApplicationNumber | 13958040 | - |
dc.identifier.patentRegistrationNumber | 9232775 | - |
dc.date.application | 2013-08-02 | - |
dc.date.registration | 2016-01-12 | - |
dc.publisher.country | US | - |
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