DC Field | Value | Language |
---|---|---|
dc.contributor.author | Kim, Ho Min | ko |
dc.contributor.author | Kim, You-Me | ko |
dc.date.accessioned | 2018-11-12T04:51:41Z | - |
dc.date.available | 2018-11-12T04:51:41Z | - |
dc.date.created | 2018-10-29 | - |
dc.date.created | 2018-10-29 | - |
dc.date.created | 2018-10-29 | - |
dc.date.created | 2018-10-29 | - |
dc.date.issued | 2018-10 | - |
dc.identifier.citation | IMMUNITY, v.49, no.4, pp.582 - 584 | - |
dc.identifier.issn | 1074-7613 | - |
dc.identifier.uri | http://hdl.handle.net/10203/246559 | - |
dc.description.abstract | Recognition of cytoplasmic lipopolysaccharide (LPS) by caspase-11 leads to pyroptosis and secretion of inflammatory mediators. In this issue of Immunity, Deng et al. (2018) report that high-mobility group box 1 (HMGB1) secreted by hepatocytes delivers extracellular LPS into the cytoplasm and mediates pyroptosis. | - |
dc.language | English | - |
dc.publisher | CELL PRESS | - |
dc.title | HMGB1: LPS Delivery Vehicle for Caspase-11-Mediated Pyroptosis | - |
dc.type | Article | - |
dc.identifier.wosid | 000447386700003 | - |
dc.identifier.scopusid | 2-s2.0-85054428143 | - |
dc.type.rims | ART | - |
dc.citation.volume | 49 | - |
dc.citation.issue | 4 | - |
dc.citation.beginningpage | 582 | - |
dc.citation.endingpage | 584 | - |
dc.citation.publicationname | IMMUNITY | - |
dc.identifier.doi | 10.1016/j.immuni.2018.09.021 | - |
dc.contributor.localauthor | Kim, Ho Min | - |
dc.contributor.localauthor | Kim, You-Me | - |
dc.description.isOpenAccess | N | - |
dc.type.journalArticle | Editorial Material | - |
dc.subject.keywordPlus | LIPOPOLYSACCHARIDE | - |
dc.subject.keywordPlus | CD14 | - |
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